High-intensity Raf signal causes cell cycle arrest mediated by p21Cip1.
Activated Raf has been linked to such opposing cellular responses as the induction of DNA synthesis and the inhibition of proliferation. However, it remains unclear how such a switch in signal specificity is regulated. We have addressed this question with a regulatable Raf-androgen receptor fusion p...
保存先:
| 出版年: | Mol Cell Biol |
|---|---|
| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Taylor & Francis
1997
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC232407/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9271434/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.17.9.5588 |
| タグ: |
タグなし, このレコードへの初めてのタグを付けませんか!
|