E7 Abolishes Raf-Induced Arrest via Mislocalization of p21(Cip1)

The cellular response to oncogenic Ras depends upon the presence or absence of cooperating mutations. In the absence of immortalizing oncogenes or genetic lesions, activation of the Ras/Raf pathway results in a p21(Cip1)-dependent cellular arrest. The human papillomavirus oncoprotein E7 transforms p...

Täydet tiedot

Bibliografiset tiedot

Julkaisussa:	Mol Cell Biol
Päätekijät:	Westbrook, Thomas F., Nguyen, Don X., Thrash, Barry R., McCance, Dennis J.
Aineistotyyppi:	Artigo
Kieli:	Inglês
Julkaistu:	Taylor & Francis 2002
Aiheet:	Transcriptional Regulation
Linkit:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC139800/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12242284/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.22.20.7041-7052.2002
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