E7 Abolishes Raf-Induced Arrest via Mislocalization of p21(Cip1)

The cellular response to oncogenic Ras depends upon the presence or absence of cooperating mutations. In the absence of immortalizing oncogenes or genetic lesions, activation of the Ras/Raf pathway results in a p21(Cip1)-dependent cellular arrest. The human papillomavirus oncoprotein E7 transforms p...

詳細記述

書誌詳細

出版年:	Mol Cell Biol
主要な著者:	Westbrook, Thomas F., Nguyen, Don X., Thrash, Barry R., McCance, Dennis J.
フォーマット:	Artigo
言語:	Inglês
出版事項:	Taylor & Francis 2002
主題:	Transcriptional Regulation
オンライン･アクセス:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC139800/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12242284/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.22.20.7041-7052.2002
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