High-intensity Raf signal causes cell cycle arrest mediated by p21Cip1.
Activated Raf has been linked to such opposing cellular responses as the induction of DNA synthesis and the inhibition of proliferation. However, it remains unclear how such a switch in signal specificity is regulated. We have addressed this question with a regulatable Raf-androgen receptor fusion p...
שמור ב:
| הוצא לאור ב: | Mol Cell Biol |
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| Principais autores: | , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Taylor & Francis
1997
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC232407/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9271434/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.17.9.5588 |
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