Breakers of advanced glycation end products restore large artery properties in experimental diabetes
Glucose and other reducing sugars react with proteins by a nonenzymatic, posttranslational modification process called nonenzymatic glycation. The formation of advanced glycation end products (AGEs) on connective tissue and matrix components accounts largely for the increase in collagen crosslinking...
Tallennettuna:
| Julkaisussa: | Proc Natl Acad Sci U S A |
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| Päätekijät: | , , , , , , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
National Academy of Sciences
1998
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC22541/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9539789/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.95.8.4630 |
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