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Dephosphorylation of eIF-2α Mediated by the γ(1)34.5 Protein of Herpes Simplex Virus Type 1 Is Required for Viral Response to Interferon but Is Not Sufficient for Efficient Viral Replication

The γ(1)34.5 protein of herpes simplex virus type 1 (HSV-1) functions to block the shutoff of protein synthesis involving double-stranded RNA-dependent protein kinase (PKR). In this process, the γ(1)34.5 protein recruits cellular protein phosphatase 1 (PP1) to form a high-molecular-weight complex th...

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Autori principali: Cheng, Guofeng, Yang, Kui, He, Bin
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society for Microbiology 2003
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC224583/
https://ncbi.nlm.nih.gov/pubmed/12941928
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.77.18.10154-10161.2003
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