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Defects in coding joint formation in vivo in developing ATM-deficient B and T lymphocytes

Ataxia-telangiectasia mutated (ATM)–deficient lymphocytes exhibit defects in coding joint formation during V(D)J recombination in vitro. Similar defects in vivo should affect both T and B cell development, yet the lymphoid phenotypes of ATM deficiency are more pronounced in the T cell compartment. I...

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Detalhes bibliográficos
Main Authors: Huang, Ching-Yu, Sharma, Girdhar G., Walker, Laura M., Bassing, Craig H., Pandita, Tej K., Sleckman, Barry P.
Formato: Artigo
Idioma:Inglês
Publicado em: The Rockefeller University Press 2007
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2118620/
https://ncbi.nlm.nih.gov/pubmed/17502661
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20061460
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