Epicutaneous antigen exposure induces a Th17 response that drives airway inflammation after inhalation challenge

Lignende værker

• Filaggrin deficient mice exhibit Th17-dominated skin inflammation and permissiveness to epicutaneous sensitization with protein antigen
  af: Oyoshi, Michiko K., et al.
  Udgivet: (2009)
• The prostaglandin D2 receptor CRTH2 is important for allergic skin inflammation following epicutaneous antigen challenge
  af: He, Rui, et al.
  Udgivet: (2010)
• Exaggerated IL-17 response to epicutaneous sensitization mediates airway inflammation in the absence of IL-4 and IL-13
  af: He, Rui, et al.
  Udgivet: (2009)
• IL-22 promotes allergic airway inflammation in epicutaneously sensitized mice
  af: Leyva-Castillo, Juan Manuel, et al.
  Udgivet: (2018)
• Epicutaneous challenge of orally immunized mice redirects antigen-specific gut-homing T cells to the skin
  af: Oyoshi, Michiko K., et al.
  Udgivet: (2011)