Hepatitis B virus replication is cell cycle independent during liver regeneration in transgenic mice.

The content of hepatitis B virus (HBV) replicative forms and HBV core protein in the liver of HBV transgenic mice is transiently reduced during massive liver regeneration following partial hepatectomy while the steady-state content of viral RNA is unchanged. This antiviral effect is triggered by int...

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Bibliografische gegevens

Gepubliceerd in:	J Virol
Hoofdauteurs:	Guidotti, L G, Matzke, B, Chisari, F V
Formaat:	Artigo
Taal:	Inglês
Gepubliceerd in:	American Society for Microbiology (ASM) 1997
Onderwerpen:	Research Article
Online toegang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC191703/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9151875/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.71.6.4804-4808.1997
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