Hepatitis B virus replication is cell cycle independent during liver regeneration in transgenic mice.
The content of hepatitis B virus (HBV) replicative forms and HBV core protein in the liver of HBV transgenic mice is transiently reduced during massive liver regeneration following partial hepatectomy while the steady-state content of viral RNA is unchanged. This antiviral effect is triggered by int...
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| Publicado no: | J Virol |
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| Principais autores: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Microbiology (ASM)
1997
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC191703/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9151875/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.71.6.4804-4808.1997 |
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