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Hepatitis B virus replication is cell cycle independent during liver regeneration in transgenic mice.

The content of hepatitis B virus (HBV) replicative forms and HBV core protein in the liver of HBV transgenic mice is transiently reduced during massive liver regeneration following partial hepatectomy while the steady-state content of viral RNA is unchanged. This antiviral effect is triggered by int...

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Bibliografiska uppgifter
Huvudupphovsmän: Guidotti, L G, Matzke, B, Chisari, F V
Materialtyp: Artigo
Språk:Inglês
Publicerad: 1997
Ämnen:
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC191703/
https://ncbi.nlm.nih.gov/pubmed/9151875
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