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Hepatitis B virus replication is cell cycle independent during liver regeneration in transgenic mice.

The content of hepatitis B virus (HBV) replicative forms and HBV core protein in the liver of HBV transgenic mice is transiently reduced during massive liver regeneration following partial hepatectomy while the steady-state content of viral RNA is unchanged. This antiviral effect is triggered by int...

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Detalhes bibliográficos
Publicado no:J Virol
Principais autores: Guidotti, L G, Matzke, B, Chisari, F V
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Microbiology (ASM) 1997
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC191703/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9151875/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.71.6.4804-4808.1997
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