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Response of human mammary epithelial cells to DNA damage induced by 4-hydroxyequilenin: lack of p53-mediated G(1) arrest

Long-term exposure to synthetic and endogenous estrogens has been associated with the development of cancer in several tissues. One potential mechanism of estrogen carcinogenesis involves catechol formation and these catechols are further oxidized to electrophilic/redox active o-quinones, which have...

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Detalles Bibliográficos
Autores principales: Cuendet, Muriel, Bolton, Judy L.
Formato: Artigo
Lenguaje:Inglês
Publicado: 2006
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC1862785/
https://ncbi.nlm.nih.gov/pubmed/16730688
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cbi.2006.04.004
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