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Response of human mammary epithelial cells to DNA damage induced by 4-hydroxyequilenin: lack of p53-mediated G(1) arrest

Long-term exposure to synthetic and endogenous estrogens has been associated with the development of cancer in several tissues. One potential mechanism of estrogen carcinogenesis involves catechol formation and these catechols are further oxidized to electrophilic/redox active o-quinones, which have...

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Détails bibliographiques
Auteurs principaux: Cuendet, Muriel, Bolton, Judy L.
Format: Artigo
Langue:Inglês
Publié: 2006
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC1862785/
https://ncbi.nlm.nih.gov/pubmed/16730688
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cbi.2006.04.004
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