Interferon-γ and Tumor Necrosis Factor-α Regulate Amyloid-β Plaque Deposition and β-Secretase Expression in Swedish Mutant APP Transgenic Mice

Podobne knjige/članki

• Overexpression of Monocyte Chemotactic Protein-1/CCL2 in β-Amyloid Precursor Protein Transgenic Mice Show Accelerated Diffuse β-Amyloid Deposition
  od: Yamamoto, Masaru, et al.
  Izdano: (2005)
• Cytokine-mediated inhibition of fibrillar amyloid-β peptide degradation by human mononuclear phagocytes
  od: Yamamoto, Masaru, et al.
  Izdano: (2008)
• CNS expression of anti-inflammatory cytokine interleukin-4 attenuates Alzheimer’s disease-like pathogenesis in APP+PS1 bigenic mice
  od: Kiyota, Tomomi, et al.
  Izdano: (2010)
• Phenolic Bis-Styrylbenzenes as β-Amyloid Binding Ligands and Free Radical Scavengers
  od: Flaherty, Daniel P., et al.
  Izdano: (2010)
• AAV1/2-mediated CNS Gene Delivery of Dominant-negative CCL2 Mutant Suppresses Gliosis, β-amyloidosis, and Learning Impairment of APP/PS1 Mice
  od: Kiyota, Tomomi, et al.
  Izdano: (2009)