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Rce1 deficiency accelerates the development of K-RAS–induced myeloproliferative disease
The RAS proteins undergo farnesylation of a carboxyl-terminal cysteine (the “C” of the carboxyl-terminal CaaX motif). After farnesylation, the 3 amino acids downstream from the farnesyl cysteine (the -aaX of the CaaX motif) are released by RAS-converting enzyme 1 (RCE1). We previously showed that in...
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| 主要な著者: | , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society of Hematology
2007
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1785091/ https://ncbi.nlm.nih.gov/pubmed/16973961 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2006-05-024752 |
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