Rce1 deficiency accelerates the development of K-RAS–induced myeloproliferative disease

The RAS proteins undergo farnesylation of a carboxyl-terminal cysteine (the “C” of the carboxyl-terminal CaaX motif). After farnesylation, the 3 amino acids downstream from the farnesyl cysteine (the -aaX of the CaaX motif) are released by RAS-converting enzyme 1 (RCE1). We previously showed that in...

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Bibliografiske detaljer
Main Authors: Wahlstrom, Annika M., Cutts, Briony A., Karlsson, Christin, Andersson, Karin M. E., Liu, Meng, Sjogren, Anna-Karin M., Swolin, Birgitta, Young, Stephen G., Bergo, Martin O.
Format: Artigo
Sprog:Inglês
Udgivet: American Society of Hematology 2007
Fag:
Neoplasia
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC1785091/
https://ncbi.nlm.nih.gov/pubmed/16973961
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2006-05-024752
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