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Effects of (−)-RO363 at human atrial β-adrenoceptor subtypes, the human cloned β(3)-adrenoceptor and rodent intestinal β(3)-adrenoceptors
1. Chronic treatment of patients with β-blockers causes atrial inotropic hyperresponsiveness through β(2)-adrenoceptors, 5-HT(4) receptors and H(2)-receptors but apparently not through β(1)-adrenoceptors despite data claiming an increased β(1)-adrenoceptor density from homogenate binding studies. We...
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| Asıl Yazarlar: | , , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
1997
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1564358/ https://ncbi.nlm.nih.gov/pubmed/9117106 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.bjp.0700850 |
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