Effects of (−)-RO363 at human atrial β-adrenoceptor subtypes, the human cloned β(3)-adrenoceptor and rodent intestinal β(3)-adrenoceptors

1. Chronic treatment of patients with β-blockers causes atrial inotropic hyperresponsiveness through β(2)-adrenoceptors, 5-HT(4) receptors and H(2)-receptors but apparently not through β(1)-adrenoceptors despite data claiming an increased β(1)-adrenoceptor density from homogenate binding studies. We...

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Hlavní autoři: Molenaar, Peter, Sarsero, Doreen, Arch, Jonathan R S, Kelly, John, Henson, Sian M, Kaumann, Alberto J
Médium: Artigo
Jazyk:Inglês
Vydáno: 1997
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Papers
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC1564358/
https://ncbi.nlm.nih.gov/pubmed/9117106
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.bjp.0700850
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