Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy

פריטים דומים

• Endocytosis machinery is required for β(1)-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes
  מאת: Morisco, Carmine, et al.
  יצא לאור: (2008)
• Mst1 inhibition rescues β(1)-adrenergic cardiomyopathy by reducing myocyte necrosis and non-myocyte apoptosis rather than myocyte apoptosis
  מאת: Lee, Grace J., et al.
  יצא לאור: (2015)
• Inhibition of endogenous thioredoxin in the heart increases oxidative stress and cardiac hypertrophy
  מאת: Yamamoto, Mitsutaka, et al.
  יצא לאור: (2003)
• The MEKK1-JNK pathway plays a protective role in pressure overload but does not mediate cardiac hypertrophy
  מאת: Sadoshima, Junichi, et al.
  יצא לאור: (2002)
• Cardiac-specific overexpression of AT1 receptor mutant lacking Gαq/Gαi coupling causes hypertrophy and bradycardia in transgenic mice
  מאת: Zhai, Peiyong, et al.
  יצא לאור: (2005)