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Atrial natriuretic peptide and oxytocin induce natriuresis by release of cGMP

Our hypothesis is that oxytocin (OT) causes natriuresis by activation of renal NO synthase that releases NO followed by cGMP that mediates the natriuresis. To test this hypothesis, an inhibitor of NO synthase, l-nitroarginine methyl ester (NAME), was injected into male rats. Blockade of NO release b...

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Bibliografiska uppgifter
Huvudupphovsmän:	Soares, T. J., Coimbra, T. M., Martins, A. R., Pereira, A. G. F., Carnio, E. C., Branco, L. G. S., Albuquerque-Araujo, W. I. C., de Nucci, G., Favaretto, A. L. V., Gutkowska, J., McCann, S. M., Antunes-Rodrigues, J.
Materialtyp:	Artigo
Språk:	Inglês
Publicerad:	The National Academy of Sciences 1999
Ämnen:	Biological Sciences
Länkar:	https://ncbi.nlm.nih.gov/pmc/articles/PMC15130/ https://ncbi.nlm.nih.gov/pubmed/9874809
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Atrial natriuretic peptide and oxytocin induce natriuresis by release of cGMP

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