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Atrial natriuretic peptide and oxytocin induce natriuresis by release of cGMP

Our hypothesis is that oxytocin (OT) causes natriuresis by activation of renal NO synthase that releases NO followed by cGMP that mediates the natriuresis. To test this hypothesis, an inhibitor of NO synthase, l-nitroarginine methyl ester (NAME), was injected into male rats. Blockade of NO release b...

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Veröffentlicht in:Proc Natl Acad Sci U S A
Hauptverfasser: Soares, T. J., Coimbra, T. M., Martins, A. R., Pereira, A. G. F., Carnio, E. C., Branco, L. G. S., Albuquerque-Araujo, W. I. C., de Nucci, G., Favaretto, A. L. V., Gutkowska, J., McCann, S. M., Antunes-Rodrigues, J.
Format: Artigo
Sprache:Inglês
Veröffentlicht: National Academy of Sciences 1999
Schlagworte:
Biological Sciences
Online-Zugang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC15130/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9874809/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.96.1.278
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