Atrial natriuretic peptide and oxytocin induce natriuresis by release of cGMP

Our hypothesis is that oxytocin (OT) causes natriuresis by activation of renal NO synthase that releases NO followed by cGMP that mediates the natriuresis. To test this hypothesis, an inhibitor of NO synthase, l-nitroarginine methyl ester (NAME), was injected into male rats. Blockade of NO release b...

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Bibliografiske detaljer

Udgivet i:	Proc Natl Acad Sci U S A
Principais autores:	Soares, T. J., Coimbra, T. M., Martins, A. R., Pereira, A. G. F., Carnio, E. C., Branco, L. G. S., Albuquerque-Araujo, W. I. C., de Nucci, G., Favaretto, A. L. V., Gutkowska, J., McCann, S. M., Antunes-Rodrigues, J.
Format:	Artigo
Sprog:	Inglês
Udgivet:	National Academy of Sciences 1999
Fag:	Biological Sciences
Online adgang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC15130/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9874809/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.96.1.278
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