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Muscle-specific inactivation of the IGF-I receptor induces compensatory hyperplasia in skeletal muscle
During the development of skeletal muscle, myoblasts withdraw from the cell cycle and differentiate into myotubes. The insulin-like growth factors IGF-I and IGF-II, through their cognate tyrosine kinase receptor (IGF-I receptor), are known to play a role in this process. After withdrawal of myoblast...
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| Autors principals: | , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society for Clinical Investigation
2002
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC150853/ https://ncbi.nlm.nih.gov/pubmed/11827994 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI13503 |
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