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A marked deficiency in circulating and renal IGF-I peptide does not inhibit compensatory renal enlargement in uninephrectomized mice
OBJECTIVE: Increase in kidney IGF-I levels due to its increased trapping from the circulation was hypothesized to be a key mediator of compensatory renal enlargement. We tested this hypothesis using genetically engineered mice with extremely low circulating IGF-I levels. DESIGN: Both IGF-I deficient...
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| Publicado no: | Growth Horm IGF Res |
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| Main Authors: | , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5488277/ https://ncbi.nlm.nih.gov/pubmed/21862442 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ghir.2011.07.008 |
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