Wordt geladen...
A NOVEL KCNJ11 MUTATION ASSOCIATED WITH CONGENITAL HYPERINSULINISM REDUCES THE INTRINSIC OPEN PROBABILITY OF β-CELL ATP-SENSITIVE POTASSIUM CHANNELS
The β-cell ATP-sensitive potassium (K(ATP)) channel controls insulin secretion by linking glucose metabolism to membrane excitability. Loss of K(ATP) channel function due to mutations in ABCC8 or KCNJ11, genes that encode the sulfonylurea receptor 1 or the inward rectifier Kir6.2 subunit of the chan...
Bewaard in:
| Hoofdauteurs: | , , , , |
|---|---|
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
2005
|
| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1479853/ https://ncbi.nlm.nih.gov/pubmed/16332676 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M511875200 |
| Tags: |
Voeg label toe
Geen labels, Wees de eerste die dit record labelt!
|