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DNA Damage Signaling and p53-dependent Senescence after Prolonged β-Interferon Stimulation

Interferons are cytokines with potent antiviral and antiproliferative activities. We report that although a transient exposure to β-interferon induces a reversible cell cycle arrest, a sustained treatment triggers a p53-dependent senescence program. β-Interferon switched on p53 in two steps. First,...

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Autors principals: Moiseeva, Olga, Mallette, Frédérick A., Mukhopadhyay, Utpal K., Moores, Adrian, Ferbeyre, Gerardo
Format: Artigo
Idioma:Inglês
Publicat: The American Society for Cell Biology 2006
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC1415317/
https://ncbi.nlm.nih.gov/pubmed/16436515
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E05-09-0858
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