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β-Arrestin 1 down-regulation after insulin treatment is associated with supersensitization of β2 adrenergic receptor Gαs signaling in 3T3-L1 adipocytes

β-Arrestin 1 is required for internalization and mitogen-activated protein (MAP) kinase activation by the β2 adrenergic receptor (β2AR). Our previous studies have shown that chronic insulin treatment down-regulates cellular β-arrestin 1 levels, leading to a marked impairment in G protein-coupled rec...

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Bibliografiset tiedot
Päätekijät: Hupfeld, Christopher J., Dalle, Stephane, Olefsky, Jerrold M.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: The National Academy of Sciences 2003
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC140914/
https://ncbi.nlm.nih.gov/pubmed/12509508
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0235674100
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