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Complex I deficiency primes Bax-dependent neuronal apoptosis through mitochondrial oxidative damage
Dysfunction of mitochondrial complex I is a feature of human neurodegenerative diseases such as Leber hereditary optic neuropathy and Parkinson's disease. This mitochondrial defect is associated with a recruitment of the mitochondrial-dependent apoptotic pathway in vivo. However, in isolated br...
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| Hlavní autoři: | , , , , , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
National Academy of Sciences
2005
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1323177/ https://ncbi.nlm.nih.gov/pubmed/16365298 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0508215102 |
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