Two novel phosphorylation sites on FKHR that are critical for its nuclear exclusion

FKHR is phosphorylated by protein kinase B (PKB) at Thr24, Ser256 and Ser319 in response to growth factors, stimulating the nuclear exit and inactivation of this transcription factor. Here we identify two further residues, Ser322 and Ser325, that become phosphorylated in insulin-like growth factor-1...

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Vydáno v:	EMBO J
Hlavní autoři:	Rena, Graham, Woods, Yvonne L., Prescott, Alan R., Peggie, Mark, Unterman, Terry G., Williams, Michayla R., Cohen, Philip
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	Nature Publishing Group 2002
Témata:	Article
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC125977/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11980723/ https://ncbi.nlm.nih.govhttps://doi.org/10.1093/emboj/21.9.2263
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