ARF Directly Binds DP1: Interaction with DP1 Coincides with the G(1) Arrest Function of ARF

The tumor suppressor ARF inhibits cell growth in response to oncogenic stress in a p53-dependent manner. Also, there is an increasing appreciation of ARF's ability to inhibit cell growth via multiple p53-independent mechanisms, including its ability to regulate the E2F pathway. We have investig...

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Autors principals: Datta, Abhishek, Sen, Jayita, Hagen, Jussara, Korgaonkar, Chandrashekhar K., Caffrey, Michael, Quelle, Dawn E., Hughes, Douglas E., Ackerson, Timothy J., Costa, Robert H., Raychaudhuri, Pradip
Format: Artigo
Idioma:Inglês
Publicat: American Society for Microbiology 2005
Matèries:
Signal Transduction
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC1234342/
https://ncbi.nlm.nih.gov/pubmed/16135794
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.25.18.8024-8036.2005
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