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ARF Directly Binds DP1: Interaction with DP1 Coincides with the G(1) Arrest Function of ARF
The tumor suppressor ARF inhibits cell growth in response to oncogenic stress in a p53-dependent manner. Also, there is an increasing appreciation of ARF's ability to inhibit cell growth via multiple p53-independent mechanisms, including its ability to regulate the E2F pathway. We have investig...
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| Hauptverfasser: | , , , , , , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
American Society for Microbiology
2005
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1234342/ https://ncbi.nlm.nih.gov/pubmed/16135794 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.25.18.8024-8036.2005 |
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