Recent developments in biomarkers in Parkinson disease

بواسطة Schapira, Anthony H.V.
منشور في 2013

Mitochondrial Contribution to Parkinson's Disease Pathogenesis

بواسطة Schapira, Anthony H. V., Gegg, Matthew
منشور في 2011

Exploring the Genotype–Phenotype Correlation in GBA-Parkinson Disease: Clinical Aspects, Biomarkers, and Potential Modifiers

بواسطة Menozzi, Elisa, Schapira, Anthony H. V.
الحاوية / القاعدة Front Neurol (2021)

Mitochondrial disorders in neurology /

بواسطة Schapira, Anthony H.V., DiMauro, S.
منشور في 1994.

Glucocerebrosidase in the pathogenesis and treatment of Parkinson disease

بواسطة Schapira, Anthony H. V., Gegg, Matthew E.
منشور في 2013

PINK1-parkin-dependent mitophagy involves ubiquitination of mitofusins 1 and 2: Implications for Parkinson disease pathogenesis

بواسطة Gegg, Matthew E., Schapira, Anthony H.V.
منشور في 2011

Mitochondrial dysfunction associated with glucocerebrosidase deficiency

بواسطة Gegg, Matthew E., Schapira, Anthony H.V.
الحاوية / القاعدة Neurobiol Dis (2016)

The relationship between glucocerebrosidase mutations and Parkinson disease

بواسطة Migdalska‐Richards, Anna, Schapira, Anthony H. V.
الحاوية / القاعدة J Neurochem (2016)

Principles of treatment in Parkinson's disease /

بواسطة Schapira, Anthony H V, Olanow, C. Warrren
منشور في 2005.

The gut-brain axis and Parkinson disease: clinical and pathogenetic relevance

بواسطة Menozzi, Elisa, Macnaughtan, Jane, Schapira, Anthony H. V.
الحاوية / القاعدة Ann Med (2021)

Glucocerebrosidase deficiency promotes release of α-synuclein fibrils from cultured neurons

بواسطة Gegg, Matthew E, Verona, Guglielmo, Schapira, Anthony H V
الحاوية / القاعدة Hum Mol Genet (2020)

Meclizine-induced enhanced glycolysis is neuroprotective in Parkinson disease cell models

بواسطة Hong, Chien Tai, Chau, Kai-Yin, Schapira, Anthony H. V.
الحاوية / القاعدة Sci Rep (2016)

The Cytomegalovirus protein pUL37×1 targets mitochondria to mediate neuroprotection

بواسطة Hong, Chien Tai, Chau, Kai-Yin, Schapira, Anthony H. V.
الحاوية / القاعدة Sci Rep (2016)

Ambroxol effects in glucocerebrosidase and α‐synuclein transgenic mice

بواسطة Migdalska‐Richards, Anna, Daly, Liam, Bezard, Erwan, Schapira, Anthony H. V.
الحاوية / القاعدة Ann Neurol (2016)

Mitochondrial and lysosomal biogenesis are activated following PINK1/parkin‐mediated mitophagy

بواسطة Ivankovic, Davor, Chau, Kai‐Yin, Schapira, Anthony H. V., Gegg, Matthew E.
الحاوية / القاعدة J Neurochem (2015)

Silencing of PINK1 Expression Affects Mitochondrial DNA and Oxidative Phosphorylation in DOPAMINERGIC Cells

بواسطة Gegg, Matthew E., Cooper, J. Mark, Schapira, Anthony H. V., Taanman, Jan-Willem
منشور في 2009

Timing of Deep Brain Stimulation in Parkinson Disease: A Need for Reappraisal?

بواسطة deSouza, Ruth-Mary, Moro, Elena, Lang, Anthony E, Schapira, Anthony H V
منشور في 2013

PINK1 disables the anti-fission machinery to segregate damaged mitochondria for mitophagy

بواسطة Pryde, Kenneth R., Smith, Heather L., Chau, Kai-Yin, Schapira, Anthony H.V.
الحاوية / القاعدة J Cell Biol (2016)

A clinical and family history study of Parkinson's disease in heterozygous glucocerebrosidase mutation carriers

بواسطة McNeill, Alisdair, Duran, Raquel, Hughes, Derralynn A, Mehta, Atul, Schapira, Anthony H V
منشور في 2012

A Human Neural Crest Stem Cell-Derived Dopaminergic Neuronal Model Recapitulates Biochemical Abnormalities in GBA1 Mutation Carriers

بواسطة Yang, Shi-Yu, Beavan, Michelle, Chau, Kai-Yin, Taanman, Jan-Willem, Schapira, Anthony H.V.
الحاوية / القاعدة Stem Cell Reports (2017)