A noninflammatory pathway for pregnancy loss: innate immune activation?

Por Salmon, Jane E.
Publicado em 2004

Autoantibody selection and production in early human life

Por Meffre, Eric, Salmon, Jane E.
Publicado em 2007

Theodore E. Woodward Award: Antiphospholipid Syndrome Revisited: A Disorder Initiated by Inflammation

Por Salmon, Jane E., Girardi, Guillermina
Publicado em 2007

Antiphospholipid antibodies and pregnancy loss: a disorder of inflammation

Por Salmon, Jane E., Girardi, Guillermina
Publicado em 2007

The Role of Complement Inhibition in Thrombotic Angiopathies and Antiphospholipid Syndrome

Por Erkan, Doruk, Salmon, Jane E.
Publicado no Turk J Haematol (2016)

Subclinical Atherosclerosis in Rheumatoid Arthritis and Systemic Lupus Erythematosus

Por Salmon, Jane E., Roman, Mary J.
Publicado em 2008

Dysregulated Complement Activation as a Common Pathway of Injury in Preeclampsia and Other Pregnancy Complications

Por Lynch, Anne M., Salmon, Jane E.
Publicado em 2010

How does IFN-α insult the vasculature? Let me count the ways

Por Kaplan, Mariana J., Salmon, Jane E.
Publicado em 2011

Are post-translational modifications of ϐ2-glycoprotein I markers for thrombotic risk? Are they triggers of autoimmunity?

Por Lockshin, Michael D., Salmon, Jane E.
Publicado em 2011

Pathogenic Role of Antiphospholipid Antibodies

Por Salmon, Jane E., de Groot, Philip G.
Publicado em 2008

Complement regulates conventional DC-mediated NK-cell activation by inducing TGF-β1 in Gr-1(+) myeloid cells

Por Qing, Xiaoping, Koo, Gloria C., Salmon, Jane E.
Publicado em 2012

Antiphospholipid Syndrome and Pregnancy: Pathogenesis to Translation

Por Abrahams, Vikki M., Chamley, Lawrence W., Salmon, Jane E.
Publicado no Arthritis Rheumatol (2017)

Excessive complement activation is associated with placental injury in patients with antiphospholipid antibodies

Por Shamonki, Jaime M., Salmon, Jane E., Hyjek, Elizabeth, Baergen, Rebecca N.
Publicado em 2007

Sex ratio of offspring born to women with systemic lupus erythematosus and rheumatoid arthritis

Por Arkema, Elizabeth V., Askling, Johan, Salmon, Jane E., Simard, Julia F.
Publicado no Arthritis Rheumatol (2016)

Complement activation induces dysregulation of angiogenic factors and causes fetal rejection and growth restriction

Por Girardi, Guillermina, Yarilin, Dmitry, Thurman, Joshua M., Holers, V. Michael, Salmon, Jane E.
Publicado em 2006

Tissue factor: a link between C5a and neutrophil activation in antiphospholipid antibody–induced fetal injury

Por Redecha, Patricia, Tilley, Rachel, Tencati, Michael, Salmon, Jane E., Kirchhofer, Daniel, Mackman, Nigel, Girardi, Guillermina
Publicado em 2007

SLE Monocytes are Less Responsive to IL-10 in the Presence of Immune Complexes

Por Yuan, Weijia, DiMartino, Stephen J., Redecha, Patricia B., Ivashkiv, Lionel B., Salmon, Jane E.
Publicado em 2011

What to Expect When Expecting With Systemic Lupus Erythematosus (SLE): A Population‐Based Study of Maternal and Fetal Outcomes in SLE and Pre‐SLE

Por Arkema, Elizabeth V., Palmsten, Kristin, Sjöwall, Christopher, Svenungsson, Elisabet, Salmon, Jane E., Simard, Julia F.
Publicado no Arthritis Care Res (Hoboken) (2016)

N-Terminal Pro-Brain Natriuretic Peptide is Associated with Cardiac but not Vascular Disease in Systemic Lupus Erythematosus Patients

Por Goldenberg, Diana, Miller, Emily, Perna, Michelle, Sattar, Naveed, Welsh, Paul, Roman, Mary J., Salmon, Jane E.
Publicado em 2012

Engraftment of PBMC from SLE and APS Donors into BALB-Rag2−/−IL2Rgc−/− Mice: a Promising Model for Studying Human Disease

Por Andrade, Danieli, Redecha, Patricia B., Vukelic, Milena, Qing, Xiaoping, Perino, Giorgio, Salmon, Jane E., Koo, Gloria C.
Publicado em 2011