Are post-translational modifications of ϐ2-glycoprotein I markers for thrombotic risk? Are they triggers of autoimmunity?

Eitemau Tebyg

• Dysbiosis May Trigger Autoimmune Diseases via Inappropriate Post-Translational Modification of Host Proteins
  gan: Lerner, Aaron, et al.
  Cyhoeddwyd: (2016)
• Archaeal S-layer glycoproteins: post-translational modification in the face of extremes
  gan: Kandiba, Lina, et al.
  Cyhoeddwyd: (2014)
• Oxidative post-translational modifications and their involvement in the pathogenesis of autoimmune diseases
  gan: Ryan, Brent J., et al.
  Cyhoeddwyd: (2014)
• Regulation of T-Cell Signaling by Post-Translational Modifications in Autoimmune Disease
  gan: Kuwabara, Taku, et al.
  Cyhoeddwyd: (2018)
• Effector-triggered post-translational modifications and their role in suppression of plant immunity
  gan: Andrew James Mark Howden, et al.
  Cyhoeddwyd: (2012-07-01)