When sparks get old

Por Melzer, Werner
Publicado em 2006

Skeletal muscle fibers: Inactivated or depleted after long depolarizations?

Por Melzer, Werner
Publicado em 2013

No voltage change at skeletal muscle SR membrane during Ca(2+) release—just Mermaids on acid

Por Melzer, Werner
Publicado no J Gen Physiol (2018)

ECC meets CEU—New focus on the backdoor for calcium ions in skeletal muscle cells

Por Melzer, Werner
Publicado no J Gen Physiol (2020)

A retrograde signal from RyR1 alters DHP receptor inactivation and limits window Ca(2+) release in muscle fibers of Y522S RyR1 knock-in mice

Por Andronache, Zoita, Hamilton, Susan L., Dirksen, Robert T., Melzer, Werner
Publicado em 2009

Calcium transients in developing mouse skeletal muscle fibres

Por Capote, Joana, Bolaños, Pura, Schuhmeier, Ralph Peter, Melzer, Werner, Caputo, Carlo
Publicado em 2005

Altered Inactivation of Ca(2+) Current and Ca(2+) Release in Mouse Muscle Fibers Deficient in the DHP receptor γ(1) subunit

Por Ursu, Daniel, Schuhmeier, Ralph Peter, Freichel, Marc, Flockerzi, Veit, Melzer, Werner
Publicado em 2004

Fast-to-Slow Transition of Skeletal Muscle Contractile Function and Corresponding Changes in Myosin Heavy and Light Chain Formation in the R6/2 Mouse Model of Huntington’s Disease...

Por Hering, Tanja, Braubach, Peter, Landwehrmeyer, G. Bernhard, Lindenberg, Katrin S., Melzer, Werner
Publicado no PLoS One (2016)

The auxiliary subunit γ(1) of the skeletal muscle L-type Ca(2+) channel is an endogenous Ca(2+) antagonist

Por Andronache, Zoita, Ursu, Daniel, Lehnert, Simone, Freichel, Marc, Flockerzi, Veit, Melzer, Werner
Publicado em 2007

The Ca(2+) influx through the mammalian skeletal muscle dihydropyridine receptor is irrelevant for muscle performance

Por Dayal, Anamika, Schrötter, Kai, Pan, Yuan, Föhr, Karl, Melzer, Werner, Grabner, Manfred
Publicado no Nat Commun (2017)

Voltage modulates halothane-triggered Ca(2+) release in malignant hyperthermia-susceptible muscle

Por Zullo, Alberto, Textor, Martin, Elischer, Philipp, Mall, Stefan, Alt, Andreas, Klingler, Werner, Melzer, Werner
Publicado no J Gen Physiol (2018)

Altered Ca(2+) signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington’s disease

Por Braubach, Peter, Orynbayev, Murat, Andronache, Zoita, Hering, Tanja, Landwehrmeyer, Georg Bernhard, Lindenberg, Katrin S., Melzer, Werner
Publicado em 2014

Functional Interaction of Ca(V) Channel Isoforms with Ryanodine Receptors Studied in Dysgenic Myotubes

Por Schuhmeier, Ralph Peter, Gouadon, Elodie, Ursu, Daniel, Kasielke, Nicole, Flucher, Bernhard E., Grabner, Manfred, Melzer, Werner
Publicado em 2005

S100A1 promotes action potential-initiated calcium release flux and force production in skeletal muscle

Por Prosser, Benjamin L., Hernández-Ochoa, Erick O., Lovering, Richard M., Andronache, Zoita, Zimmer, Danna B., Melzer, Werner, Schneider, Martin F.
Publicado em 2010

Loss of S100A1 expression leads to Ca(2+) release potentiation in mutant mice with disrupted CaM and S100A1 binding to CaMBD2 of RyR1

Por Hernández‐Ochoa, Erick O., Melville, Zephan, Vanegas, Camilo, Varney, Kristen M., Wilder, Paul T., Melzer, Werner, Weber, David J., Schneider, Martin F.
Publicado no Physiol Rep (2018)

Muscle weakness in Ryr1(I4895T/WT) knock-in mice as a result of reduced ryanodine receptor Ca(2+) ion permeation and release from the sarcoplasmic reticulum

Por Loy, Ryan E., Orynbayev, Murat, Xu, Le, Andronache, Zoita, Apostol, Simona, Zvaritch, Elena, MacLennan, David H., Meissner, Gerhard, Melzer, Werner, Dirksen, Robert T.
Publicado em 2011

Modulation of sarcoplasmic reticulum Ca(2+) release in skeletal muscle expressing ryanodine receptor impaired in regulation by calmodulin and S100A1

Por Yamaguchi, Naohiro, Prosser, Benjamin L., Ghassemi, Farshid, Xu, Le, Pasek, Daniel A., Eu, Jerry P., Hernández-Ochoa, Erick O., Cannon, Brian R., Wilder, Paul T., Lovering, Richard M., Weber, David, Melzer, Werner, Schneider, Martin F., Meissner, Gerhard
Publicado em 2011