Bone Morphogenetic Protein Signaling Modulates Myocardin Transactivation of Cardiac Genes

Por Callis, Thomas E., Cao, Dongsun, Wang, Da-Zhi
Publicado em 2005

microRNAs and muscle disorders

Por Chen, Jian-Fu, Callis, Thomas E., Wang, Da-Zhi
Publicado em 2009

MicroRNAs 1, 133, and 206: Critical factors of skeletal and cardiac muscle development, function, and disease

Por Townley-Tilson, W.H. Davin, Callis, Thomas E., Wang, Da-Zhi
Publicado em 2009

Evolving molecular diagnostics for familial cardiomyopathies: at the heart of it all

Por Callis, Thomas E, Jensen, Brian C, Weck, Karen E, Willis, Monte S
Publicado no Expert Rev Mol Diagn (2010)

miR-155 Inhibits Expression of the MEF2A Protein to Repress Skeletal Muscle Differentiation

Por Seok, Hee Young, Tatsuguchi, Mariko, Callis, Thomas E., He, Aibin, Pu, William T., Wang, Da-Zhi
Publicado em 2011

Distinguishing Hypertrophic Cardiomyopathy-Associated Mutations from Background Genetic Noise

Por Kapplinger, Jamie D., Landstrom, Andrew P., Bos, J. Martijn, Salisbury, Benjamin A., Callis, Thomas E., Ackerman, Michael J.
Publicado no J Cardiovasc Transl Res (2014)

Myocardin inhibits cellular proliferation by inhibiting NF-κB(p65)-dependent cell cycle progression

Por Tang, Ru-hang, Zheng, Xi-Long, Callis, Thomas E., Stansfield, William E., He, Jiayin, Baldwin, Albert S., Wang, Da-Zhi, Selzman, Craig H.
Publicado em 2008

The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation

Por Chen, Jian-Fu, Mandel, Elizabeth M, Thomson, J Michael, Wu, Qiulian, Callis, Thomas E, Hammond, Scott M, Conlon, Frank L, Wang, Da-Zhi
Publicado em 2005

The BMP pathway acts to directly regulate Tbx20 in the developing heart

Por Mandel, Elizabeth M., Kaltenbrun, Erin, Callis, Thomas E., Zeng, Xin-Xin I., Marques, Sara R., Yelon, Deborah, Wang, Da-Zhi, Conlon, Frank L.
Publicado em 2010

Enhancing the Predictive Power of Mutations in the C-Terminus of the KCNQ1-Encoded Kv7.1 Voltage-Gated Potassium Channel

Por Kapplinger, Jamie D., Tseng, Andrew S., Salisbury, Benjamin A., Tester, David J., Callis, Thomas E., Alders, Marielle, Wilde, Arthur A.M., Ackerman, Michael J.
Publicado no J Cardiovasc Transl Res (2015)

Expression of microRNAs is dynamically regulated during cardiomyocyte hypertrophy

Por Tatsuguchi, Mariko, Seok, Hee Young, Callis, Thomas E., Thomson, J. Michael, Chen, Jian-Fu, Newman, Martin, Rojas, Mauricio, Hammond, Scott M., Wang, Da-Zhi
Publicado em 2007

Enhanced Classification of Brugada Syndrome–Associated and Long-QT Syndrome–Associated Genetic Variants in the SCN5A-Encoded Na(v)1.5 Cardiac Sodium Channel

Por Kapplinger, Jamie D., Giudicessi, John R., Ye, Dan, Tester, David J., Callis, Thomas E., Valdivia, Carmen R., Makielski, Jonathan C., Wilde, Arthur A., Ackerman, Michael J.
Publicado no Circ Cardiovasc Genet (2015)

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice

Por Callis, Thomas E., Pandya, Kumar, Seok, Hee Young, Tang, Ru-Hang, Tatsuguchi, Mariko, Huang, Zhan-Peng, Chen, Jian-Fu, Deng, Zhongliang, Gunn, Bronwyn, Shumate, Janelle, Willis, Monte S., Selzman, Craig H., Wang, Da-Zhi
Publicado em 2009

Targeted deletion of Dicer in the heart leads to dilated cardiomyopathy and heart failure

Por Chen, Jian-Fu, Murchison, Elizabeth P., Tang, Ruhang, Callis, Thomas E., Tatsuguchi, Mariko, Deng, Zhongliang, Rojas, Mauricio, Hammond, Scott M., Schneider, Michael D., Selzman, Craig H., Meissner, Gerhard, Patterson, Cam, Hannon, Gregory J., Wang, Da-Zhi
Publicado em 2008

Distinguishing Arrhythmogenic Right Ventricular Cardiomyopathy/Dysplasia-Associated Mutations from Background Genetic Noise

Por Kapplinger, Jamie D., Landstrom, Andrew P., Salisbury, Benjamin A., Callis, Thomas E., Pollevick, Guido D., Tester, David J., Cox, Moniek G. P. J., Bhuiyan, Zahir, Bikker, Hennie, Wiesfeld, Ans C. P., Hauer, Richard N. W., van Tintelen, J. Peter, Jongbloed, Jan D. H., Calkins, Hugh, Judge, Daniel P., Wilde, Arthur A. M., Ackerman, Michael J.
Publicado no J Am Coll Cardiol (2011)

Association of arrhythmia-related genetic variants with phenotypes documented in electronic medical records

Por Van Driest, Sara L., Wells, Quinn S., Stallings, Sarah, Bush, William S., Gordon, Adam, Nickerson, Deborah A., Kim, Jerry H., Crosslin, David R., Jarvik, Gail P., Carrell, David S., Ralston, James, Larson, Eric B., Bielinski, Suzette J., Olson, Janet E., Ye, Zi, Kullo, Iftikhar J., Abul-Husn, Noura S., Scott, Stuart A., Bottinger, Erwin, Almoguera, Berta, Connolly, John, Chiavacci, Rosetta, Hakonarson, Hakon, Rasmussen-Torvik, Laura J., Pan, Vivian, Persell, Stephen D., Smith, Maureen, Chisholm, Rex L., Kitchner, Terrie E., He, Max M., Brilliant, Murray H., Wallace, John R., Doheny, Kimberly F., Shoemaker, M. Benjamin, Li, Rongling, Manolio, Teri A., Callis, Thomas E., Macaya, Daniela, Williams, Marc S., Carey, David, Kapplinger, Jamie D., Ackerman, Michael J., Ritchie, Marylyn D., Denny, Joshua C., Roden, Dan M.
Publicado no JAMA (2016)