A Novel interaction of CLN3 with nonmuscle myosin-IIB and defects in cell motility of Cln3(−/−) cells

Por Getty, Amanda L, Benedict, Jared W, Pearce, David A
Publicado em 2010

Interaction between Sdo1p and Btn1p in the Saccharomyces cerevisiae model for Batten disease

Por Vitiello, Seasson Phillips, Benedict, Jared W., Padilla-Lopez, Sergio, Pearce, David A.
Publicado em 2010

Protein Product of CLN6 Gene Responsible for Variant Late-Onset Infantile Neuronal Ceroid Lipofuscinosis Interacts with CRMP-2

Por Benedict, Jared W., Getty, Amanda L., Wishart, Thomas M., Gillingwater, Thomas H., Pearce, David A.
Publicado em 2009

Cerebellar defects in a mouse model of juvenile neuronal ceroid lipofuscinosis

Por Weimer, Jill M., Benedict, Jared W., Getty, Amanda L., Pontikis, Charlie C., Lim, Ming J., Cooper, Jonathan D., Pearce, David A.
Publicado em 2009

Visual deficits in a mouse model of Batten disease are the result of optic nerve degeneration and loss of dorsal lateral geniculate thalamic neurons

Por Weimer, Jill M., Custer, Andrew W., Benedict, Jared W., Alexander, Noreen A., Kingsley, Evan, Federoff, Howard J., Cooper, Jonathan D., Pearce, David A.
Publicado em 2006

Alterations in striatal dopamine catabolism precede loss of substantia nigra neurons in a mouse model of Juvenile Neuronal Ceroid Lipofuscinosis

Por Weimer, Jill M., Benedict, Jared W., Elshatory, Yasser M., Short, Douglas W., Ramirez-Montealegre, Denia, Ryan, Deborah A., Alexander, Noreen A., Federoff, Howard J., Cooper, Jonathan D., Pearce, David A.
Publicado no Brain Res (2007)