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A mostrar 1 - 20 resultados de 43 para a pesquisa '(( j small compared law ngai ) OR ((( a (nagyl OR natl) clamp a non ) OR ( j nay cam cam nova ))))*', tempo de pesquisa: 2.97seg Refinar resultados
Efficacy of small-volume simethicone given at least 30 min before gastroscopy
Patch-clamp recording of amino acid-activated responses in "organotypic" slice cultures.

The diabetogenic effects of glucocorticoids are more pronounced in low- than in high-insulin responders.
Active site voltage clamp fluorometry of the sodium glucose cotransporter hSGLT1

Unique properties of non-N-methyl-D-aspartate excitatory responses in cultured purkinje neurons.
Activation of heteromeric G protein-gated inward rectifier K(+) channels overexpressed by adenovirus gene transfer inhibits the excitability of hippocampal neurons

Latent N-methyl-D-aspartate receptors in the recurrent excitatory pathway between hippocampal CA1 pyramidal neurons: Ca(2+)-dependent activation by blocking A1 adenosine receptors.
PIP2 mediates functional coupling and pharmacology of neuronal KCNQ channels

Cooperativity between the orthosteric and allosteric ligand binding sites of RORγt
Citrate modulates the regulation by Zn2+ of N-methyl-D-aspartate receptor-mediated channel current and neurotransmitter release.

Construction of a high-affinity receptor site for dihydropyridine agonists and antagonists by single amino acid substitutions in a non-l-type Ca(2+) channel
Differentially synchronized spiking enables multiplexed neural coding

Purified unitary kainate/alpha-amino-3-hydroxy-5-methylisooxazole-propionate (AMPA) and kainate/AMPA/N-methyl-D-aspartate receptors with interchangeable subunits.
Nanoscale remodeling of ryanodine receptor cluster size underlies cerebral microvascular dysfunction in Duchenne muscular dystrophy

Studies on the mechanism of general anesthesia
Structure-guided examination of the mechanogating mechanism of PIEZO2

Synaptotagmin 1 oligomers clamp and regulate different modes of neurotransmitter release
Dynamic clamp constructed phase diagram for the Hodgkin and Huxley model of excitability

Somatostatin depresses excitability in neurons of the solitary tract complex through hyperpolarization and augmentation of IM, a non-inactivating voltage-dependent outward current blocked by muscarinic agonists.
Decreased muscle glucose transport/phosphorylation is an early defect in the pathogenesis of non-insulin-dependent diabetes mellitus.

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