NOD Mice Are Defective in Proteasome Production and Activation of NF-κB
The nonobese diabetic (NOD) mouse is an animal model of human type I diabetes with a strong genetic component that maps to the major histocompatibility complex (MHC) of the genome. We have identified in NOD lymphocytes a specific proteasome defect that results from the lack of the LMP2 subunit. The...
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| I publikationen: | Mol Cell Biol |
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| Huvudupphov: | , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Utgiven: |
Taylor & Francis
1999
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC85003/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10567588/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.19.12.8646 |
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