טוען...
Striatal GABAergic interneuron dysfunction in the Q175 mouse model of Huntington’s disease
The pathological hallmark of Huntington’s disease (HD) is the massive loss of striatal and cortical neurons. Until recently, it was believed that striatal interneurons were spared from degeneration. This view has changed after the demonstration that parvalbumin (PV)-expressing interneurons also are...
שמור ב:
| הוצא לאור ב: | Eur J Neurosci |
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| Main Authors: | , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2018
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8320683/ https://ncbi.nlm.nih.gov/pubmed/30472747 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/ejn.14283 |
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