p53 Deacetylation Alleviates Sepsis-Induced Acute Kidney Injury by Promoting Autophagy

Recent studies have shown that autophagy upregulation can attenuate sepsis-induced acute kidney injury (SAKI). The tumor suppressor p53 has emerged as an autophagy regulator in various forms of acute kidney injury (AKI). Our previous studies showed that p53 acetylation exacerbated hemorrhagic shock-...

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Bibliographische Detailangaben
Veröffentlicht in:Front Immunol
Hauptverfasser: Sun, Maomao, Li, Jiaxin, Mao, Liangfeng, Wu, Jie, Deng, Zhiya, He, Man, An, Sheng, Zeng, Zhenhua, Huang, Qiaobing, Chen, Zhongqing
Format: Artigo
Sprache:Inglês
Veröffentlicht: Frontiers Media S.A. 2021
Schlagworte:
Immunology
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC8318785/
https://ncbi.nlm.nih.gov/pubmed/34335587
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fimmu.2021.685523
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