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Membrane Interactions Accelerate the Self-Aggregation of Huntingtin Exon 1 Fragments in a Polyglutamine Length-Dependent Manner

The accumulation of aggregated protein is a typical hallmark of many human neurodegenerative disorders, including polyglutamine-related diseases such as chorea Huntington. Misfolding of the amyloidogenic proteins gives rise to self-assembled complexes and fibres. The huntingtin protein is characteri...

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Foilsithe in:Int J Mol Sci
Main Authors: Marquette, Arnaud, Aisenbrey, Christopher, Bechinger, Burkhard
Formáid: Artigo
Teanga:Inglês
Foilsithe: MDPI 2021
Ábhair:
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC8268948/
https://ncbi.nlm.nih.gov/pubmed/34201610
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/ijms22136725
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