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An Arrhythmic Mutation E7K Facilitates TRPM4 Channel Activation via Enhanced PIP(2) Interaction
A Ca(2+)-activated monovalent cation-selective TRPM4 channel is abundantly expressed in the heart. Recently, a single gain-of-function mutation identified in the distal N-terminus of the human TRPM4 channel (Glu(5) to Lys(5); E7K) was found to be arrhythmogenic because of enhanced cell membrane expr...
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| Publicat a: | Cells |
|---|---|
| Autors principals: | , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
MDPI
2021
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8146980/ https://ncbi.nlm.nih.gov/pubmed/33922380 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/cells10050983 |
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