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CD4(+) T cells require Ikaros to inhibit their differentiation toward a pathogenic cell fate
The production of proinflammatory cytokines, particularly granulocyte-macrophage colony-stimulating factor (GM-CSF), by pathogenic CD4(+) T cells is central for mediating tissue injury in inflammatory and autoimmune diseases. However, the factors regulating the T cell pathogenic gene expression prog...
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| Publicat a: | Proc Natl Acad Sci U S A |
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| Autors principals: | , , , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
National Academy of Sciences
2021
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8092604/ https://ncbi.nlm.nih.gov/pubmed/33893236 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.2023172118 |
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