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Cyclin M2 (CNNM2) knockout mice show mild hypomagnesaemia and developmental defects
Patients with mutations in Cyclin M2 (CNNM2) suffer from hypomagnesaemia, seizures, and intellectual disability. Although the molecular function of CNNM2 is under debate, the protein is considered essential for renal Mg(2+) reabsorption. Here, we used a Cnnm2 knock out mouse model, generated by CRIS...
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| Gepubliceerd in: | Sci Rep |
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| Hoofdauteurs: | , , , , , , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Nature Publishing Group UK
2021
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8050252/ https://ncbi.nlm.nih.gov/pubmed/33859252 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-021-87548-6 |
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