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The Thrombopoietin Receptor, MPL, Is a Therapeutic Target of Opportunity in the MPN

The myeloproliferative neoplasms, polycythemia vera, essential thrombocytosis and primary myelofibrosis share driver mutations that either activate the thrombopoietin receptor, MPL, or indirectly activate it through mutations in the gene for JAK2, its cognate tyrosine kinase. Paradoxically, although...

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Foilsithe in:Front Oncol
Main Authors: Spivak, Jerry L., Moliterno, Alison R.
Formáid: Artigo
Teanga:Inglês
Foilsithe: Frontiers Media S.A. 2021
Ábhair:
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC7987816/
https://ncbi.nlm.nih.gov/pubmed/33777803
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fonc.2021.641613
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