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Elevated post-ischemic ubiquitination results from suppression of deubiquitinase activity and not proteasome inhibition
Cerebral ischemia–reperfusion increases intraneuronal levels of ubiquitinated proteins, but the factors driving ubiquitination and whether it results from altered proteostasis remain unclear. To address these questions, we used in vivo and in vitro models of cerebral ischemia–reperfusion, in which h...
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| Publicado no: | Cell Mol Life Sci |
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| Main Authors: | , , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Springer International Publishing
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7933347/ https://ncbi.nlm.nih.gov/pubmed/32889561 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00018-020-03625-5 |
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