טוען...
DNA polymerase ι compensates for Fanconi anemia pathway deficiency by countering DNA replication stress
Fanconi anemia (FA) is caused by defects in cellular responses to DNA crosslinking damage and replication stress. Given the constant occurrence of endogenous DNA damage and replication fork stress, it is unclear why complete deletion of FA genes does not have a major impact on cell proliferation and...
שמור ב:
| הוצא לאור ב: | Proc Natl Acad Sci U S A |
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| Main Authors: | , , , , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
National Academy of Sciences
2020
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7777187/ https://ncbi.nlm.nih.gov/pubmed/33376220 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.2008821117 |
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