DNA polymerase ι compensates for Fanconi anemia pathway deficiency by countering DNA replication stress

Fanconi anemia (FA) is caused by defects in cellular responses to DNA crosslinking damage and replication stress. Given the constant occurrence of endogenous DNA damage and replication fork stress, it is unclear why complete deletion of FA genes does not have a major impact on cell proliferation and...

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Dades bibliogràfiques
Publicat a:Proc Natl Acad Sci U S A
Autors principals: Wang, Rui, Lenoir, Walter F., Wang, Chao, Su, Dan, McLaughlin, Megan, Hu, Qianghua, Shen, Xi, Tian, Yanyan, Klages-Mundt, Naeh, Lynn, Erica, Wood, Richard D., Chen, Junjie, Hart, Traver, Li, Lei
Format: Artigo
Idioma:Inglês
Publicat: National Academy of Sciences 2020
Matèries:
Biological Sciences
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC7777187/
https://ncbi.nlm.nih.gov/pubmed/33376220
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.2008821117
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