DNA polymerase ι compensates for Fanconi anemia pathway deficiency by countering DNA replication stress

Fanconi anemia (FA) is caused by defects in cellular responses to DNA crosslinking damage and replication stress. Given the constant occurrence of endogenous DNA damage and replication fork stress, it is unclear why complete deletion of FA genes does not have a major impact on cell proliferation and...

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Bibliografiset tiedot
Julkaisussa:Proc Natl Acad Sci U S A
Päätekijät: Wang, Rui, Lenoir, Walter F., Wang, Chao, Su, Dan, McLaughlin, Megan, Hu, Qianghua, Shen, Xi, Tian, Yanyan, Klages-Mundt, Naeh, Lynn, Erica, Wood, Richard D., Chen, Junjie, Hart, Traver, Li, Lei
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: National Academy of Sciences 2020
Aiheet:
Biological Sciences
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC7777187/
https://ncbi.nlm.nih.gov/pubmed/33376220
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.2008821117
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