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DNA polymerase ι compensates for Fanconi anemia pathway deficiency by countering DNA replication stress
Fanconi anemia (FA) is caused by defects in cellular responses to DNA crosslinking damage and replication stress. Given the constant occurrence of endogenous DNA damage and replication fork stress, it is unclear why complete deletion of FA genes does not have a major impact on cell proliferation and...
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| Publicado en: | Proc Natl Acad Sci U S A |
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| Autores principales: | , , , , , , , , , , , , , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
National Academy of Sciences
2020
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7777187/ https://ncbi.nlm.nih.gov/pubmed/33376220 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.2008821117 |
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