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Tyrosine Phosphorylation of the K(v)2.1 Channel Contributes to Injury in Brain Ischemia
In brain ischemia, oxidative stress induces neuronal apoptosis, which is mediated by increased activity of the voltage-gated K(+) channel K(v)2.1 and results in an efflux of intracellular K(+). The molecular mechanisms underlying the regulation of K(v)2.1 and its activity during brain ischemia are n...
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| Publicado no: | Int J Mol Sci |
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| Main Authors: | , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
MDPI
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7765428/ https://ncbi.nlm.nih.gov/pubmed/33333928 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/ijms21249538 |
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